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Ginkgo Leaf (Premium Grade) 24% Flavonglycosides\6% Triterpenes Powdered Extract (Ginkgo biloba; Bai Guo Ye) 25 kg (55 lbs): Q

Ginkgo Leaf (Premium Grade) 24% Flavonglycosides\6% Triterpenes Powdered Extract (Ginkgo biloba; Bai Guo Ye) 25 kg (55 lbs): Q

Can be used as an infusion, decoction, extract, tincture, food additive and supplement. Medicinal use of Ginkgo leaf tea is ancient in Chinese herbal medicine. It helps clear the mind and improve the power of concentration. Modern clinical trials have demonstrated the usefulness of ginkgo in the treatment of Alzheimer�??s disease, age-related cognitive decline and depression. Ginkgo's flavone glycosides and terpene lactones do indeed increase circulation to the brain and other parts of the body as well as exerting a protective action on nerve cells. The unique terpene lactone components found in ginkgo are known as ginkgolides and bilobalide. Ginkgolides apparently improve circulation and inhibit platelet-activating factor. Bilobalide protects the cells of the nervous system. Recent animal studies indicate that bilobalide may help regenerate damaged nerve cells. One double blind study found that ginkgo could help people with macular degeneration, an oxidation-related disorder causing decreased or lost vision. Ginkgo�??s antioxidant activity in the brain and central nervous system may help prevent age-related declines in brain function. The brain and central nervous system are particularly susceptible to free radical attack. Free radical damage, that is, oxidation damage, in the brain is widely accepted as being a contributing factor in many disorders associated with aging, including Alzheimer�??s disease. The 1997 Commission E on Phytotherapy and Herbal Substances of the German Federal Institute for Drugs recommends a dry extract from the dried leaf of Ginkgo biloba for '(a) symptomatic treatment of disturbed performance in organic brain syndrome within the regimen of a therapeutic concept in cases of demential syndromes with the following principal symptoms: Memory deficits, disturbances in concentration, depressive emotional condition, dizziness, tinnitus, and headache.' '(b) Improvement of pain-free walking distance in peripheral arterial occlusive disease in Stage II of Fontaine (intermittent claudication) in a regimen of physical therapeutic measures, in particular walking exercise. (c) Vertigo and tinnitus (ringing in the ear) of vascular and involutional origin.' 'The following pharmacological effects have been established experimentally: Improvement of hypoxic tolerance, particularly in the cerebral tissue. Inhibition of the development of traumatically or toxically induced cerebral edema, and acceleration of its regression. Reduction of retinal edema and of cellular lesions in the retina. Inhibition in age-related reduction of muscarinergic cholinoceptors and 2-adrenoceptors as well as stimulation of choline uptake in the hippocampus. Increased memory performance and learning capacity. Improvement in the compensation of disturbed equilibrium. Improvement of blood flow, particularly in the region of microcirculation. Improvement of the rheological properties of the blood. Inactivation of toxi

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